By Hugh Aldersey-Williams

ISBN-10: 0393240479

ISBN-13: 9780393240474

"A impressive, organ-by-organ trip in the course of the physique eclectic. . . impossible to resist [and] impressive."—John J. Ross, Wall highway JournalThe human physique is the main fraught and interesting, talked-about and taboo, distinct but common truth of our lives. it's the concept for paintings, the topic of technology, and the resource of a few of the best tales ever advised. In Anatomies, acclaimed writer of Periodic stories Hugh Aldersey-Williams brings his interesting combination of technological know-how, background, and tradition to endure in this richest of subjects.

In an enticing narrative that levels from historic physique artwork to cosmetic surgery at the present time and from head to toe, Aldersey-Williams explores the corporeal mysteries that make us human: Why are a few humans left-handed and a few blue-eyed? what's the humorous bone, besides? Why perform a little cultures contemplate the guts because the seat of our souls and passions, whereas others position it within the liver?

A journalist with a knack for telling a narrative, Aldersey-Williams takes half in a drawing category, attends the dissection of a human physique, and visits the doctor's workplace and the morgue. yet Anatomies attracts not only on scientific technology and Aldersey-Williams's reporting. It attracts additionally at the works of philosophers, writers, and artists from all through historical past. Aldersey-Williams delves into our shared cultural heritage—Shakespeare to Frankenstein, Rembrandt to 2001: an area Odyssey—to exhibit how attitudes towards the human physique are as assorted as human historical past, as he explains the origins and legacy of tattooing, shrunken heads, bloodletting, fingerprinting, X-rays, and more.

From Adam's rib to van Gogh's ear to Einstein's mind, Anatomies is a treasure trove of bizarre evidence and tales and an excellent embodiment of what Aristotle wrote greater than millennia in the past: "The human physique is greater than the sum of its parts."

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"A magnificent, organ-by-organ trip during the physique eclectic. . . impossible to resist [and] awesome. "—John J. Ross, Wall road JournalThe human physique is the main fraught and engaging, talked-about and taboo, specified but common truth of our lives. it's the suggestion for paintings, the topic of technology, and the resource of a few of the best tales ever instructed.

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Injection of very small doses of DT into highly susceptible animals by the intradermal route causes dermonecrosis. The ability of circulating anti-DT antibodies to neutralize this dermonecrotic response to DT was the basis for the Schick test, introduced in 1913, as a means to distinguish between individuals who are susceptible to diphtheria and those with acquired immunity to DT who are resistant to diphtheria. Studies in the 1950s showed that small amounts of DT were able to kill a variety of eukaryotic cell lines derived from susceptible animals (Lennox and Kaplan 1957; Placido Sousa and Evans 1957), and inhibition of protein synthesis was shown to be the first manifestation of toxicity in HeLa cells exposed to DT (Strauss and Hendee 1959).

Diphtheriae C4(β) or C7(β) lysogens, the β-prophages can be induced to enter the lytic cycle of phage replication by exposing the lysogenic bacteria to ultraviolet light (Groman and Lockart 1953; Barksdale and Pappenheimer 1954; Matsuda and Barksdale 1967). W. K. Holmes available about the process of maturation and release of mature virions during lytic replication of phage β. A variety of genetic and molecular studies suggest that the genome of phage β integrates into the chromosome of C. diphtheriae in a manner similar to integration of the λ-phage genome into the chromosome of E.

Diphtheriae and phage conversion of DT production, and summarizes the biology of temperate corynephages. It discusses the structure and function of DT and describes the role of DtxR as the primary global regulator of iron-dependent gene expression in C. diphtheriae. Although tox is a bacteriophage gene, its evolution as a DtxR-repressible gene within the DtxR regulon couples its expression to scarcity of intracellular iron and the presence of DtxR in its inactive apo-DtxR form. Because C. diphtheriae will predictably encounter limited bioavailability of iron in its human host, this regulatory circuit assures that DT, a critically important virulence factor, will be produced by C.

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